Hubert Czerniak - Tajemnicze związki chemiczne? Jak oddziałują na nasze zdrowie? / Wodorowęglany

Czerniak frames this as a natural, cheap alternative to sports supplements. He notes that reduced acidity directly decreases breathlessness (zadyszka) during training, and that the sodium/potassium form also covers electrolyte losses. He links the pH shift to the release of free fatty acids from adipose tissue, enabling fat-burning even at moderate intensities. Visceral fat, the dangerous 'oponka', is specifically targeted.

Czerniak describes the mechanism: during intense exercise, incomplete glucose burning produces lactic acid, causing muscle soreness. By pre-loading with bicarbonate (HCO3-), you increase the blood’s buffering capacity, which means less acidity, less pain, and longer endurance. He also claims this alkalizing effect aids in mobilizing and burning visceral fat, the 'muffin top' linked to cardiovascular disease. The protocol includes mixing the bicarbonate with an acid (vinegar, lemon, vitamin C) to create a fizzy reaction outside the body, preventing gas in the stomach. He emphasizes that this also replenishes sodium and potassium lost in sweat, addressing electrolyte needs.

Czerniak relates that Simoncini, faced with a hopeless case of bronchial cancer, applied a 20% sodium bicarbonate solution directly to the bronchi and achieved cures. He links this to bioresonance testing where patients show fungal signatures like Aspergillus niger or Candida albicans, suggesting a pre-neoplastic state. Simoncini was prosecuted and received a prison sentence, which Czerniak criticizes with irony: 'Jakaż ta Temida jest kochająca tylko czy pacjentów' (What a loving Themis, but does she love the patients?). He also notes that in hospitals, bicarbonate is given intravenously to nearly dead patients to allow them to tolerate chemotherapy, implying a broader but underutilized role.

Czerniak explains that giving bicarbonate is analogous to immobilizing a broken leg to facilitate healing. By providing the buffer externally, the kidneys’ workload decreases, preserving residual function. This insight stems from a physiological understanding where the kidney is both the source and victim of acid stress.

Czerniak emphasizes that acidemia impairs insulin receptor signaling; by normalizing pH, bicarbonate restores insulin sensitivity only where it was pathologically depressed. In healthy people, no extra sensitization occurs, so blood sugar does not drop dangerously. He stresses the dangerous nature of hypoglycemia versus hyperglycemia, noting that a person who faints is given glucose intravenously because prolonged hypoglycemia can cause severe brain damage, while even 500 mg% glucose is not immediately life-threatening.

Czerniak explains that a large majority of heartburn cases are actually caused by low stomach acid due to chronic acid suppression, not high acid. When such a patient takes bicarbonate, they feel temporary relief but perpetuate a cycle of ever-lower pH. Eventually the stomach loses its protective acidic environment (pH should be 1.2), allowing swallowed bacteria to survive, colonize the small intestine, and cause SIBO and other dysbioses. He laments that during gastroscopy, measuring gastric pH is no longer done, even though that was the original purpose of the test. He estimates true hyperacidity requiring acid blockers is about 0.1% of prescriptions.